First report on global fallout 236U and uranium atom ratios in soils from Hunan Province, China

Publication date: February 2019

Source: Journal of Environmental Radioactivity, Volume 197

Author(s): Yang Shao, Guosheng Yang, Diandou Xu, Masatoshi Yamada, Hirofumi Tazoe, Min Luo, Hangxin Cheng, Ke Yang, Lingling Ma

Abstract

More nuclear power plants continue to be built in China. Due to its long half-life, radiotoxicity and potential application as an environmental tracer, ²³⁶U is one of the most important artificial radionuclides deserving more study since activity data are important for risk assessment. However, the ultra-trace activity of ²³⁶U and its dilution by natural uranium isotopes make it difficult to distinguish its sources and there are only limited global fallout ²³⁶U data for present in Chinese environmental samples. In order to understand the background levels for uranium isotopes, especially ²³⁶U, and clarify their sources, inductively coupled plasma tandem mass spectrometry (ICP-MS/MS) was applied to analyze uranium isotopes in 48 soil samples from Hunan Province, China. The ²³⁴U, ²³⁵U, ²³⁸U and ²³⁶U concentrations were measured as 9.91–33.7, 0.312–1.43, 6.63–28.7 Bq kg⁻¹ and (1.61–21.3) × 10⁷ atoms g⁻¹, while, the ²³⁶U/²³⁸U, ²³⁴U/²³⁸U and ²³⁵U/²³⁸U atom ratios were (0.470–4.91) × 10⁻⁸, (5.10–9.31) × 10⁻⁵, and (7.11–7.82) × 10⁻³, respectively. The uranium isotopic fractionation may be due to irrigation of the agricultural lands where the samples were collected. Considering the facts that neither previous nuclear tests nor nuclear accidents had occurred in Hunan Province and the present ²³⁶U/²³⁸U atom ratios were included in the range of global fallout values in other areas, it may be concluded that ²³⁶U in soils from Hunan Province is mainly from global fallout. To the best of the authors' knowledge, the presence of global fallout ²³⁶U in soil samples from China has been confirmed for the first time, and these values may be useful as background data for risk assessment in the future.

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Acculturation and Postpartum Depression Among Immigrant Women of Arabic Descent

Abstract

Acculturation has been related to risk of postpartum depression (PPD) among immigrant women globally. The purpose of this study was to examine the relationship between acculturation and PPD symptoms among U.S. immigrant women of Arabic descent. A cross-sectional study was conducted with 115 postpartum immigrant women of Arabic descent. Women completed questionnaires including measures of acculturation [attraction to Arabic culture (AArC), attraction to American culture (AAmC), marginalization] and PPD symptoms (Edinburgh Postnatal Depression Scale—EPDS) between 1 and 12 months postpartum. Twenty-five percent of women (n = 29) had EPDS scores ≥ 10 that represent PPD symptoms. Women with higher marginalization reported more PPD symptoms (r = .25, p = .008). None of the acculturation factors correlated with PPD symptoms after adjustment for maternal sociodemographic and health characteristics. Higher education (p = .001), lower gestational age at birth (p < .05), and antenatal anxiety (p < .05) were correlated with PPD symptoms in multivariate analyses. Health care providers should identify and assess immigrant women of Arabic descent for antenatal anxiety as this may identify women at risk for development of PPD symptoms. Future studies need to examine acculturation in relation to mental health among immigrant women of Arabic descent.

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Hepatocellular carcinoma screening is associated with increased survival of patients with cirrhosis

Clinical Gastroenterology and Hepatology

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Increased mortality among patients with vs without cirrhosis and autoimmune hepatitis

Clinical Gastroenterology and Hepatology

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Mesomelia‐synostoses syndrome: Description of a patient presenting a monoallelic expression of SULF1 without alterations in the SLCOA1 gene

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Analyses of LMNA -negative juvenile progeroid cases confirms biallelic POLR3A mutations in Wiedemann–Rautenstrauch-like syndrome and expands the phenotypic spectrum of PYCR1 mutations

Abstract

Juvenile segmental progeroid syndromes are rare, heterogeneous disorders characterized by signs of premature aging affecting more than one tissue or organ starting in childhood. Hutchinson–Gilford progeria syndrome (HGPS), caused by a recurrent de novo synonymous LMNA mutation resulting in aberrant splicing and generation of a mutant product called progerin, is a prototypical example of such disorders. Here, we performed a joint collaborative study using massively parallel sequencing and targeted Sanger sequencing, aimed at delineating the underlying genetic cause of 14 previously undiagnosed, clinically heterogeneous, non-LMNA-associated juvenile progeroid patients. The molecular diagnosis was achieved in 11 of 14 cases (~ 79%). Furthermore, we firmly establish biallelic mutations in POLR3A as the genetic cause of a recognizable, neonatal, Wiedemann–Rautenstrauch-like progeroid syndrome. Thus, we suggest that POLR3A mutations are causal for a portion of under-diagnosed early-onset segmental progeroid syndromes. We additionally expand the clinical spectrum associated with PYCR1 mutations by showing that they can somewhat resemble HGPS in the first year of life. Moreover, our results lead to clinical reclassification in one single case. Our data emphasize the complex genetic and clinical heterogeneity underlying progeroid disorders.

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Comment on “Race/Ethnicity and Sex Both Affect Opioid Administration in the Emergency Room”

No abstract available

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