Nature Reviews Genetics 17, 375 (2016). doi:10.1038/nrg.2016.71
Author: Linda Koch
Researchers have elucidated the pathogenic mechanism underlying the histone H3 lysine-36-to-methionine (H3K36M) mutation, which is a frequent acquired mutation in children with chondroblastoma, a type of bone cancer. The H3K36M mutation disrupted the expression of genes involved in the differentiation of mesenchymal progenitor cells (MPCs)
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